heath and nutrition

Beyond antibiotics: “PPMOs” offer new approach to bacterial infection

CORVALLIS, Ore. – Researchers at Oregon State University and other institutions today announced the successful use of a new type of antibacterial agent called a PPMO, which appears to function as well or better than an antibiotic, but may be more precise and also solve problems with antibiotic resistance.

In animal studies, one form of PPMO showed significant control of two strains of Acinetobacter, a group of bacteria of global concern that has caused significant mortality among military personnel serving in Middle East combat.

The new PPMOs offer a fundamentally different attack on bacterial infection, researchers say.

They specifically target the underlying genes of a bacterium, whereas conventional antibiotics just disrupt its cellular function and often have broader, unwanted impacts. As they are further developed, PPMOs should offer a completely different and more precise approach to managing bacterial infection, or conceptually almost any disease that has an underlying genetic component.

The findings were published today in the Journal of Infectious Diseases, by researchers from OSU, the University of Texas Southwestern Medical Center, and Sarepta, Inc., a Corvallis, Ore., firm.

“The mechanism that PPMOs use to kill bacteria is revolutionary,” said Bruce Geller, a professor of microbiology in the OSU College of Science and lead author on the study. “They can be synthesized to target almost any gene, and in that way avoid the development of antibiotic resistance and the negative impacts sometimes associated with broad-spectrum antibiotics.

“Molecular medicine,” Geller said, “is the way of the future.”

PPMO stands for a peptide-conjugated phosphorodiamidate morpholino oligomer – a synthetic analog of DNA or RNA that has the ability to silence the expression of specific genes. Compared to conventional antibiotics, which are often found in nature, PPMOs are completely synthesized in the laboratory with a specific genetic target in mind.

In animal laboratory tests against A. baumannii, one of the most dangerous Acinetobacter strains, PPMOs were far more powerful than some conventional antibiotics like ampicillin, and comparable to the strongest antibiotics available today. They were also effective in cases where the bacteria were resistant to antibiotics.

PPMOs have not yet been tested in humans. However, their basic chemical structure, the PMO, has been extensively tested in humans and found safe. Although the addition of the peptide to the PPMO poses an uncertain risk of toxicity, the potency of PPMOs reduces the risk while greatly improving delivery of the PMOs into bacterial cells, Geller said.

Geller said research is being done with Acinetobacter in part because this pathogen has become a huge global problem, and is often spread in hospitals. It can cause respiratory infection, sepsis, and is a special concern to anyone whose immune system is compromised. Wounds in military battle conditions have led to numerous cases in veterans, and A. baumannii is now resistant to many antibiotics. “Urgent new approaches to therapeutics are needed,” the scientists said in their report.

Continued research and eventually human clinical trials will be required before the new compounds are available for health care, the researchers said. This and continued studies have been supported by the National Institutes of Health, the other collaborators and the N.L. Tartar fund.


Editor’s Note: A scanning electron microscope image of A. baumannii is available online (please provide image credit as indicated at web site): http://bit.ly/GztejR

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Bruce Geller, 541-737-1845

Innovative approach could ultimately end deadly disease of sleeping sickness

CORVALLIS, Ore. – A tag team of two bacteria, one of them genetically modified, has a good chance to reduce or even eliminate the deadly disease African trypanosomiasis, or sleeping sickness, researchers at Oregon State University conclude in a recent mathematical modeling study.

African trypanosomiasis, caused by a parasite carried by the tsetse fly, infects 30,000 people in sub-Saharan Africa each year and is almost always fatal without treatment. In a 2008 epidemic, 48,000 people died.

In this research, scientists evaluated the potential for success of a new approach to combat the disease – creating a genetically modified version of the Sodalis bacteria commonly found in the gut of the flies that carry the disease, and using different bacteria called Wolbachia to drive the GMO version of Sodalis into fly populations.

When that’s done, the GMO version of Sodalis would kill the disease-causing trypanosome parasite. According to the analysis published in PLOS Neglected Tropical Diseases, researchers say this should work – and could offer a model system for other tropical, insect-carried diseases of even greater importance, including dengue fever and malaria.

“There are a few ‘ifs’ necessary for this to succeed, but none of them look like an obstacle that could not be overcome,” said Jan Medlock, an assistant professor in the OSU Department of Biomedical Sciences, and lead author on the new report.

“If everything goes right, and we are optimistic that it will, this could be enormously important,” Medlock said. “There’s a potential here to completely solve this disease that has killed many thousands of people, and open new approaches to dealing with even more serious diseases such as malaria.”

Some of the “ifs” include: the transgenic Sodalis has to be reasonably effective at blocking the parasite, at or above a level of about 85 percent; the Wolbachia bacteria, which has some effect on the health of flies affected with it, must not kill too many of them; and the target species of fly has to be a majority of the tsetse flies in the areas being treated.

The research shows that dealing with all of those obstacles should be possible. If so, this might spell the end of a tropical disease that has plagued humans for hundreds, possibly thousands of years. African trypanosomiasis causes serious mental and physical deterioration – including the altered sleep patterns that give the disease its name – and is fatal without treatment. It’s still difficult to treat, and neurologic damage is permanent.

Past efforts to control the disease, including insect traps, insecticide spraying, and use of sterile insects have been of some value, but only in limited areas and the effects were not permanent.

The strength of the new approach, researchers say, is that once the process begins it should spread and be self-sustaining - it should not be necessary to repeatedly take action in the huge geographic areas of Africa. Due to some genetic manipulation, the flies carrying the Wolbachia bacteria should naturally increase their populations and have an inherent survival advantage over conventional tsetse flies.

As the flies carrying transgenic bacteria continue to dominate and their populations spread, trypanosomiasis should fairly rapidly disappear. Whether the mechanism of control could wane in effectiveness over time is an issue that requires further study, scientists said.

Work has begun on the GMO version of Sodalis that has the capability to resist trypanosomes . It’s not yet finalized, Medlock said, but it should be possible, and when complete, the bacteria will be very specific to tsetse flies.

Medlock, an expert in modeling the transmission of various diseases – including human influenza – says the analysis is clear that this approach has significant promise of success. Because of the relatively low infectiousness of the parasite and the ability of Wolbachia to drive the resistance genes, no part of the system has to be 100 percent perfect in order to ultimately achieve near eradication of this disease, he said.

Accomplishing a similar goal with diseases such as malaria may be more difficult, he said, because that disease historically has shown a remarkable ability to mutate and overcome many of the approaches used to attack it. However, at least some near-term gains may be possible, he said.

Collaborators on this study included scientists from the OSU College of Veterinary Medicine and the Yale School of Public Health. It was supported by the National Institutes of Health and the Miriam Weston Trust.

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Jan Medlock, 541-737-6874

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Tsetse fly

Tsetse fly

California’s new mental health system helps people live independently

CORVALLIS, Ore. – A new analysis by Oregon State University researchers of California’s mental health system finds that comprehensive, community-based mental health programs are helping people with serious mental illness transition to independent living.

Published in the October issue of the American Journal of Public Health, this study has important implications for the way that states finance and deliver mental health programs, and speaks to the effectiveness of well-funded, comprehensive community programs.

In November of 2004, California voters passed the Mental Health Services Act, which allocated more than $3 billion for comprehensive community mental health programs, known as Full Service Partnerships (FSP). While community-based, these programs are different from usual mental health services programs in most states because they provides a more intensive level of care and a broader range of mental health services and supports, such as medication management, crisis intervention, case management and peer support.

It also provides services such as food, housing, respite care and treatment for co-occurring disorders, such as substance abuse.

“We found that these programs promoted independent living in the community among people who had serious mental illness but had not been served or underserved previously,” said Jangho Yoon, an assistant professor of health policy and health economist in OSU’s College of Public Health and Human Sciences and lead author of the study. “Overall, it reduced their chance of living on the street or being incarcerated in jails and prisons.”

The researchers looked at data from 43 of California’s 53 counties, resulting in a sample of 9,208 adults over the course of four years. They found that participants who stayed enrolled in the program continuously, without interruption, were 13.5 percent more likely to successfully transition to independent living.

However, they found that non-white patients were less likely to live independently, and more likely to end up in jail or homeless.

“Although FSPs represent the most well-funded comprehensive community-based programs in the country, they are still community programs and therefore program participation is voluntary,” Yoon said.  “My guess is that minorities may not benefit fully from these programs in their communities possibly due to greater stigma, and less family/social supports. But it needs further investigation.”

Patients with schizophrenia and bipolar disorders were also less likely to benefit from the community programs, because of the nature and severity of their mental health issues.

Yoon is an expert on health management policy, specifically policy around the area of mental health. He said other states haven’t followed California’s lead, in part because of the cost of such extensive programming. Yoon said some of the funding made possible by the federal Patient Protection and Affordable Care Act, which includes $460 million for community mental health services for states to use, may help other states to create similar programs.

“Nobody would disagree that the public mental health system has historically been under-funded in the U.S.,” he said. “The message for other states is clear: investment in well-funded, recovery-oriented, comprehensive community mental health programs clearly improves lives of people with serious mental illness, and may also save money from reduced dependency and incarcerations in this population.”

Tim Bruckner of the University of California, Irvine, and Timothy Brown of the University of California, Berkeley, contributed to this study, which was jointly funded by the California Department of Mental Health and the California Health Care Foundation.

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Jangho Yoon, 541-737-3839

Gut microbes closely linked to range of health issues

CORVALLIS, Ore. –A new understanding of the essential role of gut microbes in the immune system may hold the key to dealing with some of the more significant health problems facing people in the world today, Oregon State University researchers say in a new analysis.

Problems ranging from autoimmune disease to clinical depression and simple obesity may in fact be linked to immune dysfunction that begins with a “failure to communicate” in the human gut, the scientists say. Health care of the future may include personalized diagnosis of an individual’s “microbiome” to determine what prebiotics or probiotics are needed to provide balance.

Appropriate sanitation such as clean water and sewers are good. But some erroneous lessons in health care may need to be unlearned – leaving behind the fear of dirt, the love of antimicrobial cleansers, and the outdated notion that an antibiotic is always a good idea. We live in a world of “germs” and many of them are good for us.

“Asked about their immune system, most people might think of white blood cells, lymph glands or vaccines,” said Dr. Natalia Shulzhenko, author of a new report in Clinical Reviews in Allergy and Immunology, and assistant professor and physician in the OSU Department of Biomedical Sciences. “They would be surprised that’s not where most of the action is. Our intestines contain more immune cells than the entire rest of our body.

“The human gut plays a huge role in immune function,” Shulzhenko said. “This is little appreciated by people who think its only role is digestion. The combined number of genes in the microbiota genome is 150 times larger than the person in which they reside. They do help us digest food, but they do a lot more than that.”

An emerging theory of disease, Shulzhenko said, is a disruption in the “crosstalk” between the microbes in the human gut and other cells involved in the immune system and metabolic processes.

“In a healthy person, these microbes in the gut stimulate the immune system as needed, and it in turn talks back,” Shulzhenko said. “There’s an increasing disruption of these microbes from modern lifestyle, diet, overuse of antibiotics and other issues. With that disruption, the conversation is breaking down.”

An explosion of research in the field of genomic sequencing is for the first time allowing researchers to understand some of this conversation and appreciate its significance, Shulzhenko said. The results are surprising, with links that lead to a range of diseases, including celiac disease and inflammatory bowel disease. Obesity may be related. And some studies have found relevance to depression, late-onset autism, allergies, asthma and cancer.

In the new review, researchers analyzed how microbe dysfunction can sometimes result in malabsorption and diarrhea, which affects tens of millions of children worldwide and is often not cured merely by better nutrition. In contrast, a high-fat diet may cause the gut microbes to quickly adapt to and prefer these foods, leading to increased lipid absorption and weight gain.

The chronic inflammation linked to most of the diseases that kill people in the developed world today – heart disease, cancer, diabetes – may begin with dysfunctional gut microbiota.

Understanding these processes is a first step to addressing them, Shulzhenko said. Once researchers have a better idea of what constitutes healthy microbiota in the gut, they may be able to personalize therapies to restore that balance. It should also be possible to identify and use new types of probiotics to mitigate the impact of antibiotics, when such drugs are necessary and must be used.

Such approaches are “an exciting target for therapeutic interventions” to treat health problems in the future, the researchers concluded.

The study, supported by OSU, included researchers from both the College of Veterinary Medicine and the College of Pharmacy.

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Dr. Natalia Shulzhenko, 541-737-1051

Working longer may lead to a longer life, new OSU research shows

CORVALLIS, Ore. – Working past age 65 could lead to longer life, while retiring early may be a risk factor for dying earlier, a new study from Oregon State University indicates.

The researchers found that healthy adults who retired one year past age 65 had an 11 percent lower risk of death from all causes, even when taking into account demographic, lifestyle and health issues. Adults who described themselves as unhealthy were also likely to live longer if they kept working, the findings showed, which indicates that factors beyond health may affect post-retirement mortality.

“It may not apply to everybody, but we think work brings people a lot of economic and social benefits that could impact the length of their lives,” said Chenkai Wu, the lead author of the study. He conducted the research as part of his master’s thesis at OSU, where he is now a doctoral student in the College of Public Health and Human Sciences.

The findings were published recently in the Journal of Epidemiology and Community Health. Co-authors include Associate Professor Robert Stawski and Assistant Professor Michelle Odden of OSU and Gwenith Fisher of Colorado State University. The research was supported by a grant from the National Institute on Aging.

The research was the basis for Wu’s master’s thesis in human development and family science; he’s now pursuing a doctorate in epidemiology.

Wu took an interest in the effects of retirement on health in part because of China’s mandatory laws, which are often debated. Retirement age is also an issue for debate elsewhere around the world, including the United States, he said.

“Most research in this area has focused on the economic impacts of delaying retirement. I thought it might be good to look at the health impacts,” Wu said. “People in the U.S. have more flexibility about when they retire compared to other countries, so it made sense to look at data from the U.S.”

Wu examined data collected from 1992 through 2010 through the Healthy Retirement Study, a long-term study of U.S. adults led by the University of Michigan and funded by the National Institute on Aging. Of the more than 12,000 initial participants in the study, Wu narrowed his focus to 2,956 people who began the study in 1992 and had retired by the end of the study period in 2010. 

Poor health is one reason people retire early and also can lead to earlier death, so researchers wanted to find a way to mitigate a potential bias in that regard.

To do so, they divided the group into unhealthy retirees, or those who indicated that health was a factor in their decision to retire – and healthy retirees, who indicated health was not a factor. About two-thirds of the group fell into the healthy category, while a third were in the unhealthy category.

During the study period, about 12 percent of the healthy and 25.6 percent of the unhealthy retirees died. Healthy retirees who worked a year longer had an 11 percent lower risk of mortality, while unhealthy retirees who worked a year longer had a 9 percent lower mortality risk. Working a year longer had a positive impact on the study participants’ mortality rate regardless of their health status.

“The healthy group is generally more advantaged in terms of education, wealth, health behaviors and lifestyle, but taking all of those issues into account, the pattern still remained,” said Stawski, senior author of the paper. “The findings seem to indicate that people who remain active and engaged gain a benefit from that.”

Additional research is needed to better understand the links between work and health, the researchers said. As people get older their physical health and cognitive function are likely to decline, which could affect both their ability to work and their longevity.

“This is just the tip of the iceberg,” Stawski said. “We see the relationship between work and longevity, but we don’t know everything about people’s lives, health and well-being after retirement that could be influencing their longevity.”

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Chenkai Wu, wuche@oregonstate.edu; Robert Stawski, robert.stawski@oregonstate.edu, 541-737-9052

OSU study: Silicone breast implants may absorb harmful pollutants from body tissues

CORVALLIS, Ore. – Silicone breast implants may absorb environmental pollutants from surrounding tissues and possibly reduce their concentrations within the body, according to a new study by Oregon State University.

OSU environmental chemist Kim Anderson and her colleagues found that silicone implants are a “sink” for environmental chemicals that build up in the fatty tissues of humans and animals. Over time they can become a long-term record of a person’s exposure to environmental toxins.

The study, which appeared in the journal Environmental International, was partly inspired by recent studies showing that silicone implants may reduce risk of breast cancer by as much as 50 percent, Anderson said. “That research piqued our interest in looking at implants as a potential sink for contaminants,” she said. She cautioned that her findings don’t prove that breast implants protect against cancer or any other disease.

In a two-part experiment, Anderson, a professor in OSU's College of Agricultural Sciences, and her colleagues screened eight discarded silicone breast implants—surgically removed from women for medical or personal reasons—for some 1,400 environmental chemicals. For controls, they also screened unused silicone “sizers,” used in fitting breast implants or prostheses.

The implants contained 14 common compounds used in foods and personal-care products, commercial and industrial products, and pesticides. The most common one detected was caffeine, found in all eight samples. Next was p,p’-DDE, a suspected cancer-causing agent that results from the breakdown of the now-banned insecticide DDT. It was found in five of the samples.

Nine of the chemicals showed up in only one sample. That was not surprising, said Anderson, because individual exposure to environmental chemicals varies widely depending on where a person lives and what kind of work he or she does. “People are exposed to different chemicals in Burns, Oregon, than in Portland,” she said, “and there are differences even between neighborhoods in Portland.”

In the second phase of the study, to determine how silicone implants respond to a known chemical exposure, the researchers surgically implanted tiny silicone disks into anesthetized laboratory mice. The mice received as much silicone, in proportion to their body mass, as would be used in a range of typical human breast implants or reconstructions.

The mice then were given injections of two compounds: p,p’-DDE and also PCB 118, which belongs to a class of once widely used industrial chemicals and is also a probable carcinogen. These two substances are known to accumulate in fatty tissues. A group of control mice received surgical implants but no chemicals; another control group received chemicals and surgery but no implants.

After nine days, the researchers analyzed the silicone that had been implanted in the mice along with the surrounding fatty tissue. They found that the silicon and the tissue contained both chemicals. This, said Anderson, indicated that the chemicals had passed from the tissue into the silicone until the concentrations may have reached a balance.

Silicone is known to absorb organic-based pollutants in much the same way human cells do. “Silicone is lipophilic—meaning it loves fat,” Anderson explained. “Our bodies’ cells are also lipophilic. In order for a chemical to do us harm, it has to cross our cell barriers. So silicone is a good surrogate for an organism’s cell.”

Anderson’s lab pioneered the use of silicone wristbands as passive samplers of environmental pollution in air and water. “However, those environments are hydrophilic—water-loving—which is the opposite of lipophilic,” she said. “We wanted to demonstrate that silicone would also pull contaminants out of a fat-rich environment.”

Anderson added that discarded breast implants could be a gold mine for public-health researchers. “Tens of thousands of implants are removed from women every year, and they’re typically burned as waste,” she said. “Instead, they could be an important resource for quantifying the types and amounts of environmental chemicals absorbed by the human body and for assessing long-term toxic exposure.”

The research was funded by the National Institute of Environmental Health Sciences and OSU's Food Safety and Environmental Stewardship Program.

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Kim Anderson, 541-737-8501, kim.anderson@oregonstate.edu

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Silicone breast implants may pick up chemical pollution from the body's tissues. Researcher Kim Anderson, environmental chemist at Oregon State University, wears a silicone pollution-sampling wristband. Photo by Stephen Ward.

Kim Anderson

Doctor, investigative journalist to speak at OSU

CORVALLIS, Ore. – Dr. Richard Besser, a physician, former administrator for the Centers for Disease Control and Prevention, and nationally-known investigative journalist will speak at Oregon State University on Monday, April 13, as part of the Provost’s Lecture Series.

Besser will present “A View From Both Sides of the Camera: Using Television to Promote Public Health.” The lecture will begin at 7:30 p.m. in Austin Auditorium of the LaSells Stewart Center, 875 S.W. 26th St., on the Corvallis campus. It is free and open to the public.

Besser has covered major medical news stories around the world for ABC, including several recent trips to Liberia to cover the Ebola outbreak. At the CDC, Besser was director of the Coordinating Office for Terrorism Preparedness and Emergency Response. His previous medical work included the epidemiology of food-borne diseases and pediatric tuberculosis.

His reporting has received numerous awards, including an Emmy nomination in 2011 for a piece on umbilical cord blood banking. He received two Peabody Awards as part of his work at ABC, and published his first book, “Tell Me the Truth, Doctor: Easy-to-Understand Answers to Your Most Confusing and Critical Health Questions,” in 2013.

Creating healthy people, a healthy planet and a healthy economy are top priorities for OSU, which has the state’s first accredited school of public health. For more on health-based research at the university, see the winter edition of Terra Research Magazine.

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Shelly Signs, 541-737-0724; shelly.signs@oregonstate.edu

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Richard Besser Dr. Richard Besser

Low vitamin D levels and depression linked in young women, new OSU study shows

CORVALLIS, Ore. – A new study from Oregon State University suggests there is a relationship between low levels of vitamin D and depression in otherwise healthy young women.

OSU researchers found that young women with lower levels of vitamin D were more likely to have clinically significant depressive symptoms over the course of a five-week study, lead author David Kerr said. The results were consistent even when researchers took into account other possible explanations, such as time of year, exercise and time spent outside.

“Depression has multiple, powerful causes and if vitamin D is part of the picture, it is just a small part,” said Kerr, an associate professor in the School of Psychological Science at OSU. “But given how many people are affected by depression, any little inroad we can find could have an important impact on public health.”

The findings were published recently in the journal Psychiatry Research. Co-authors are Sarina Saturn of the School of Psychological Science; Balz Frei and Adrian Gombart of OSU’s Linus Pauling Institute; David Zava of ZRT Laboratory and Walter Piper, a former OSU student now at New York University.

Vitamin D is an essential nutrient for bone health and muscle function. Deficiency has been associated with impaired immune function, some forms of cancer and cardiovascular disease, said Gombart, an associate professor of biochemistry and biophysics, principal investigator with the Linus Pauling Institute and international expert on vitamin D and the immune response.

People create their own vitamin D when their skin is exposed to sunlight. When sun is scarce in the winter, people can take a supplement, but vitamin D also is found in some foods, including milk that is fortified with it, Gombart said. The recommended daily allowance of vitamin D is 600 IU per day. There is no established level of vitamin D sufficiency for mental health.

The new study was prompted in part because there is a widely held belief that vitamin D and depression are connected, but there is not actually much scientific research out there to support the belief, Kerr said.

“I think people hear that vitamin D and depression can change with the seasons, so it is natural for them to assume the two are connected,” he said.

According to Kerr and his colleagues, a lot of past research has actually found no association between the two, but much of that research has been based on much older adults or special medical populations.

Kerr’s study focused on young women in the Pacific Northwest because they are at risk of both depression and vitamin D insufficiency. Past research found that 25 percent of American women experience clinical depression at some point in their lives, compared to 16 percent of men, for example.

OSU researchers recruited 185 college students, all women ages 18-25, to participate in the study at different times during the school year. Vitamin D levels were measured from blood samples and participants completed a depression symptom survey each week for five weeks.

Many women in the study had vitamin D levels considered insufficient for good health, and the rates were much higher among women of color, with 61 percent of women of color recording insufficient levels, compared to 35 percent of other women. In addition, more than a third of the participants reported clinically significant depressive symptoms each week over the course of the study.

“It may surprise people that so many apparently healthy young women are experiencing these health risks,” Kerr said.

As expected, the women’s vitamin D levels depended on the time of year, with levels dropping during the fall, at their lowest in winter, and rising in the spring. Depression did not show as a clear pattern, prompting Kerr to conclude that links between vitamin D deficiency and seasonal depression should be studied in larger groups of at-risk individuals.

Researchers say the study does not conclusively show that low vitamin D levels cause depression. A clinical trial examining whether vitamin D supplements might help prevent or relieve depression is the logical next step to understanding the link between the two, Kerr said.

OSU researchers already have begun a follow-up study on vitamin D deficiency in women of color. In the meantime, researchers encourage those at risk of vitamin D deficiency to speak with their doctor about taking a supplement.

“Vitamin D supplements are inexpensive and readily available.” Kerr said. “They certainly shouldn’t be considered as alternatives to the treatments known to be effective for depression, but they are good for overall health.”

The research was supported by grants from the Good Samaritan Hospital Foundation’s John C. Erkkila Endowment for Health and Human Performance and the National Institute of Environmental Health Sciences.


About the Linus Pauling Institute:  The Linus Pauling Institute at OSU is a world leader in the study of micronutrients and their role in promoting optimum health or preventing and treating disease. Major areas of research include heart disease, cancer, aging and neurodegenerative disease.

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David Kerr, 541-737-1364, david.kerr@oregonstate.edu

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Students at Oregon State University enjoy a sunny winter day on the Corvallis campus.


OSU celebrates National Nutrition Month with March 4 event

CORVALLIS, Ore. – Oregon State University’s Nutrition and Dietetics Club is celebrating National Nutrition Month on Wednesday, March 4, with an event in the Memorial Union quad from 10 a.m. to 2:30 p.m.

Its theme of “Bite Into a Healthy Lifestyle” encourages everyone to adopt eating plans focused on making informed food choices and promoting overall health. The event will feature games, prizes, free food, and tips on how to stay healthy from guests representing Bob’s Red Mill, Trader Joes, Pacific Fruit Company, Food@OSU and more.

Activities will focus around vitamins, mineral and fiber content in food, and helping students, faculty and staff learn more about what makes a healthy, balanced meal. There will be free cookbooks for the first 200 participants, a competition to win a bullet blender, and other prizes.

“Eating a healthy, well-balanced meal is crucial to success in the rest of your life, including your academic success,” said Jessica Hummel, vice president of OSU’s Nutrition and Dietetics Club. “We want students and staff to stop in and learn some fun facts about food, and maybe look at the way they eat in a new way.”

As part of this public education campaign, the academy’s National Nutrition Month website includes a variety of helpful tips, games, promotional tools and nutrition education resources, all designed to spread the message of good nutrition.  

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OSU Be Well Walk & Run on Oct. 11

CORVALLIS, Ore. – Oregon State University is holding the fourth annual Be Well Walk & Run on Oct. 11 in the Memorial Union Quad. The event is free and open to everyone.

This year’s event includes a five-kilometer running course as well as a one-mile walking course (5k Map / 1-Mile Map). The scenic route will wind participants throughout OSU’s campus, highlighting picturesque buildings and spaces. Costumes are encouraged. 

The event will feature activity stations in the Memorial Union Quad to engage participants in learning about the Healthy Campus Initiative, including physical activity, stress management, nutrition and a smoke-free campus.

Participants can register as individuals or as part of a group.  Early registrants will receive a free 2013 Be Well Walk & Run t-shirt. To register, go to http://bit.ly/19Zo7Rk. The run starts at 3:30 p.m. in the quad, check in begins at 3 p.m.

For more information, go to http://oregonstate.edu/bewell

Accommodations for disabilities may be made by calling Joe Schaffer, 541-737-4884.

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Lisa Hoogesteger, 541-737-3343

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