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Table S1

From: S. B. Prusiner (1998) Prions. Proc. Natl. Acad. Sci. USA 95: 13363-13383 (html) (pdf)

Table 1.   The prion diseases

Disease Host Mechanism of pathogenesis

Kuru Fore people Infection through ritualistic cannibalism
iCJD Humans Infection from prion-contaminated HGH, dura mater grafts, etc.
vCJD Humans Infection from bovine prions?
fCJD Humans Germ-line mutations in PrP gene
GSS Humans Germ-line mutations in PrP gene
FFI Humans Germ-line mutation in PrP gene (D178N, M129)
sCJD Humans Somatic mutation or spontaneous conversion of PrPC into PrPSc?
FSI Humans Somatic mutation or spontaneous conversion of PrPC into PrPSc?
Scrapie Sheep Infection in genetically susceptible sheep
BSE Cattle Infection with prion-contaminated MBM
TME Mink Infection with prions from sheep or cattle
CWD Mule deer, elk Unknown
FSE Cats Infection with prion-contaminated bovine tissues or MBM
Exotic ungulate encephalopathy Greater kudu, nyala, oryx Infection with prion-contaminated MBM

iCJD, iatrogenic CJD; vCJD, variant CJD; fCJD, familial CJD; sCJD, sporadic CJD; GSS, Gerstmann-Sträussler-Sheinker disease; FFI, fatal familial insomnia; FSI, fatal sporadic insomnia; BSE, bovine spongiform encephalopathy; TME, transmissible mink encephalopathy; CWD, chronic wasting disease; FSE, feline spongiform encephalopathy; HGH, human growth hormone; MBM, meat and bone meal.

Note: iatrogenic = caused by medical treatment.